Autologous Stem Cell Transplant for IgM Related AL Amyloidosis

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Autologous Stem Cell Transplant for AL Amyloidosis

AL amyloidosis is caused by clonal plasma cells that produce immunoglobulin light chains which misfold and get deposited as amyloid fibrils. Therapy directed against the plasma cell clone leads to clinical benefit. Melphalan and corticosteroids have been the mainstay of treatment for a number of years and the recent availability of other effective agents (IMiDs and proteasome inhibitors) has in...

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Sequential heart and autologous stem cell transplantation for systemic AL amyloidosis.

Extensive cardiac amyloid deposition in systemic AL amyloidosis is associated with a grave prognosis. Heart transplantation is rarely performed because of the systemic and progressive nature of the disease. Patients with severe cardiac amyloid infiltration are ineligible for the preferred treatment of melphalan chemotherapy with stem cell transplantation (SCT) rescue because of the high risk fo...

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Outcome of autologous stem cell transplantation for AL amyloidosis in the UK.

High-dose chemotherapy with autologous stem cell transplantation (SCT) is widely used as a treatment for systemic AL amyloidosis, but its efficacy has not been proved and it has substantial toxicity in this setting. We report here the outcome of 92 patients evaluated at the UK National Amyloidosis Centre who underwent SCT for AL amyloidosis between 1994 and 2004 in various British centres. Medi...

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Autologous stem cell transplantation for primary systemic amyloidosis

High-dose melphalan with autologous blood stem cell transplantation (SCT) can reverse the disease process in selected patients with primary systemic amyloidosis (AL); however, SCT for AL remains controversial because of the treatmentrelated mortality in patients with cardiac and multisystem organ involvement. In this review, we briefly discuss recent advances in AL, such as the free lightchain ...

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ژورنال

عنوان ژورنال: Biology of Blood and Marrow Transplantation

سال: 2019

ISSN: 1083-8791

DOI: 10.1016/j.bbmt.2018.12.798